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Cialis vs Viagra What's the difference between them - Daily News. Cialis and Viagra are both PDE5 inhibitors used for the treatment of erectile dysfunction (ED). Both only work when a man is sexually aroused. Cialis lasts for around 36 hours compared with only 4 to 5 hours for Viagra. Food does not affect the activity of Cialis, whereas food may decrease the effectiveness of Viagra. Cialis can be used once daily, at a smaller dosage, and is also effective at treating benign prostatic hyperplasia (BPH). Dosage of Cialis may need to be reduced in men with liver or kidney problems and risk of interactions may be higher than with Viagra.
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My favorite, clinically proven formula for naturally increasing your testosterone and sex drive: ►►http://drsam.co/yt/EDCure =============================================== 3 Powerful ED Drugs - Pros, Cons & Warnings =============================================== Today I’m going to discuss popular medications for Erectile Dysfunction (ED), pros, cons and important health warnings. I’ll quickly cover how they work, how you can make them work BETTER and lastly, natural alternatives. PDE-5 (Phosphodiesterase type 5) Inhibitors So the first type of drugs I’ll discuss, also happens to be the most popular and these are PDE-5 inhibitors, such as Viagra (sildenafil), Cialis (tadalafil) and Levitra (vardenafil). Viagra works the fastest, in about 30 minutes and lasts about 4 hours. While Cialis is the slowest -- takes about an hour or two to start working, but lasts about 2 days. Thus, known as the “weekender” pill. The “Negative” The biggest problem with these drugs is they do NOTHING for improving your sex drive and libido. Taking these drugs alone will not cause an erection. They must be taken in combination with some form of sexual stimulation. Another common problem with these drugs is side-effects such as low blood pressure, which is the original REASON these drugs were made. In fact, “erections” are a side-benefit of these drugs. There are other problems such as vision, sinus and headaches. Lastly, the lower your testosterone levels - your primary “sex drive” hormone - the LESS effective these drugs are. In fact, this is the primary reason they don’t work in about 25% of users, especially “older” guys who need it the most … Because they have “low” testosterone levels. However, there’s a way to make these drugs work better and I’ll show you how in a minute. Penile Injections Next is one of the least popular and that’s injections into your penis. A typical one is Caverject, which is a prostaglandin (PGE-1). Similar to the PDE-5 drugs, the shot increases blood flow to your penis. The results are instant and you do NOT need any kind of arousal for your penis to get hard. Unfortunately, not too many guys want to put a needle into their penis. And if you don’t do it correctly or take too much, your erection will just not go away for a long time and can actually cause tissue damage. This is best used when nothing else has worked and typically, for guys who have blood vessel problems. Increasing Testosterone Levels My personal favorite, healthiest and most long-term and “permanent” solution to erectile dysfunction is increasing your testosterone levels. The main benefit here, as stated earlier, is that testosterone is your primary “libido” hormone - in both men and women. It improves BOTH your physical and emotional sex drive. Higher testosterone also improves blood flow and nitric oxide levels, similar to Viagra. Higher testosterone also reduces many diseases (heart, diabetes, etc.) and cancers. YES - testosterone reduces cancer, it’s the higher ESTROGEN levels that cause cancer in both men and women. If you want to feel and look younger, testosterone is the primary way to do it. How To Increase Your Testosterone Levels? Now, there are 2 ways to increase testosterone: Method One For Higher Testosterone The first is “Exogenously” - which means from “external factors”, such as testosterone injections, creams, pellets, pills, etc. The second “negative” conversion is testosterone to DHT - the hair loss and prostate cancer hormone. Unfortunately, 99% of doctors are CLUELESS about the endocrine/hormonal system and thus, taking testosterone will end up causing MORE harm than good in the long-run. Oh, one other negative side-effect -- your testicles will shrink! But don’t worry, nothing will happen to your penis. Method Two For Higher Testosterone The second way to increase your testosterone is “Endogenously” - which means from “internal” factors. And this is done through eating specific foods, doing certain exercises and taking special herbs, vitamins and minerals. This is the BEST way to increase your testosterone because it’s natural, totally safe and long-term. Click here for a proven, 100% natural solution: ►http://drsam.co/yt/EDCure Or you can watch other videos related to erectile dysfunction: https://www.youtube.com/watch?v=Y_z5fqph5YU https://www.youtube.com/watch?v=fGfazrFHi70 https://www.youtube.com/watch?v=eT8_UoaqTC4 https://www.youtube.com/watch?v=AbNyVIqZ36s ========= Thank you for watching. Please feel free to comment, like or share with your friends. Subscribe to Dr.Sam Robbins's official Youtube channel http://drsam.co/yt/subscribe Like us on Facebook https://www.facebook.com/DrSamRobbins Visit Dr.Sam Robbins's blog for more information on your health! http://www.drsamrobbins.com/ ========== Thanks DrSamRobbins
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A longstanding challenge many urologists have faced, has been the treatment of erectile dysfunction (ED). Years ago medications for ED treatment, such as Viagra, came on the market which made it easier for patients suffering from erectile dysfunction to find relief. However, Viagra is one of the most short acting erectile dysfunction treatments and generally lasts approximately 6 to 8 hours. Years later, a medication came to the market by the name of Cialis, which generally lasts between 24 and 36 hours. Levitra, another erectile dysfunction medication, lasts approximately 18 hours. Most patients are surprised when they realize Cialis lasts up to 36 hours, Viagra lasts 6 to 8 hours or Levitra can last 18 hours. It is a common misconception that patients believe during this period of time they will have an erection the entire time. This is false! Although when the patient takes the medication, it circulates in the body, the patient is generally in the flaccid state. It is during any sexual stimulation or intimacy when an erection can be easily achieved and maintained. It is important to remember to always use your medication under the supervision of your physician and be sure that you are aware of the side effects of the medication before you take it.
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Shortness of breath? Learn more and get a better lung disease treatment plan with top experts. Visit our Online Expert Centers at https://diagnosticdetectives.com/expert-centers/ Exclusive interview / Dr. Anton Titov MD and Professor Dr. Aaron Waxman MD, leading pulmonary arterial hypertension, right heart failure and lung disease expert. Find out exact cause if you have shortness of breath - get a precise diagnosis and best treatment for shortness of breath root cause. - Pulmonary arterial hypertension treatment. You mentioned phosphodiesterase type 5 inhibitors. They are also used for erectile disfunction. Common medications are viagra, sildenafil, and there are other medications of that class. So they're also used to treat pulmonary arterial hypertension. What is a typical way how they are used for pulmonary arterial hypertension? Are they effective? There is probably a different dosing schedule of those medications? So phosphodiesterase 5 inhibitors are in the nitric oxide pathway. And nitric oxide response, which really is our most potent vasodilator, works through cyclic GMP. So all the phosphodiesterase 5 inhibitors do is keep that cyclic GMP around longer by preventing its breakdown. And within the same path now we also have a medication called Riociguat, which is a soluble guanylate cyclase stimulator. So it actually increases the amount of available cyclic GMP and works in parallel with nitric oxide or independently of nitric oxide in pulmonary arterial hypertension. These patients generally have inadequate NO production. So all of these drugs are vasodilators. There's probably some added benefit, although we don't fully understand yet how, as far as remodeling - both myocardial remodeling and vascular remodeling. But there's no way to know which patients are going to get the most benefit from each individual drug. But I would say from an efficacy standpoint they're all pretty equivalent in pulmonary arterial hypertension. We don't combine them, but as individual drugs they're all pretty equivalent. - Are they used early in a therapy of pulmonary arterial hypertension, or when the first line therapy fails? - No, they probably are now first line therapy. Certainly sildenafil and tadalafil are considered first-line therapy, because they're generally very well tolerated and they're easy to use and they've been around for a long time in pulmonary arterial hypertension. I think Riociguatis a little more complicated to use because there's titration involved and it has a little more broader FDA approval targeting. It includes chronic thromboembolic disease but otherwise it's probably equally effective in pulmonary arterial hypertension. - Is the typical dosage [of PDE5 inhibitors in PAH] continuous or daily? Is it a smaller dose to compare with doses used for erectile disfunction indications? - When we did the clinical trials in pulmonary arterial hypertension, we studied sildenafil at 20 milligrams, 40 milligrams and 80 milligrams, at 3 times per day. So the usual dose for erectile dysfunction is 25, 50 and 75 mg, and a 100 mg, "when you need it". So certainly during the clinical trial, before the drug was approved, we were using Viagra at those doses. So I think there's room for dose adjustment. The only dose that was approved was 20 milligrams three times a day. So from an insurance standpoint we might have a little difficulty changing dose. But we often do titrate the dose, and Tadalafilwe use it at 40 milligrams once a day. - Every day? So with this frequent and fairly large dosing for pulmonary arterial hypertension treatment, as far as the cardiovascular side effects or other side effects, have you seen any major side effects? It's interesting, because the reason we went to these drugs are because phosphodiesterase 5 is predominantly localized to the vessels of the lung and the vessels of the penis. So we actually don't see a huge incidence of cardiovascular side effects. There are some patients whose blood pressure may be a little soft [low] with these drugs. But most patients with pulmonary arterial hypertension tolerate it quite well. I think the most important side effect that we see is headache, and it's not a bad headache. But it can be a nuisance. But it's usually readily responsive to simple things like Tylenol and aspirin or a non-steroidal anti-inflammatory medications.
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